On the contrary, combination-exposed offspring demonstrated a gain of H3K27me3 peaks (potential suppression of function) compared to the diabetes-only exposed group for adenylate kinase 3 (Ak3, 1.95 fold, FDR = 0.09) and signal transducer and activator of transcription 5B (Stat5b, 3.76 fold, FDR = 0.09). This evidence concerns the gene AK3 and diabetes mellitus.