SRA-like non-coding RNA (SLNCR1) was identified as a mediator of melanoma invasion.[75] SLNCR1 interacts with both the androgen receptor (AR) and the brain-specific homeobox protein 3A (BRN3A) and increases melanoma invasion by upregulating MMP9.[75] SLNCR1 is also upregulated in NSCLC.[76] The lncRNA growth arrest specific transcript 5 (GAS5) also alters the melanoma cell invasion through interaction with matrix metalloproteinases. This evidence concerns the gene AR and non-small cell lung carcinoma.