BMI1, a member of the PRC1 family, may be specifically and directly induced in response to H3K27me3; therefore, the correlation of BMI1 expression with the expression of PRC2 markers and MALAT1 could be an indirect consequence of the sequential processes of PRC pathway activation and chromatin remodeling in T cell lymphoma, without direct binding between BMI1 and MALAT1. This evidence concerns the gene PRC1 and T-cell non-Hodgkin lymphoma.