Analysis of protein expression revealed that the expression of Bcl-2, Bax, cyt-c, AIF, caspase-3, caspase-9, Endo-G and Apaf-1 was significantly increased, prompting mitochondrial apoptosis, which plays a major role in adenomyosis after UAO treatment, and inducing irreversible apoptosis in many cells, which is consistent with the mechanism shown in elsewhere [15]. The gene discussed is BCL2; the disease is adenomyosis.