Different studies have demonstrated the protective effect of HT by (i) preventing LDL oxidation; (ii) inhibiting platelet aggregation; (iii) attenuating mitochondrial abnormalities, with HFD-induced metabolic syndrome prevention [19], and (iv) producing anti-inflammatory effects in association with a decreased activity of the enzymes cyclooxygenase 1 (COX1) and COX2 [20]. This evidence concerns the gene PTGS1 and metabolic syndrome.