The protective effects of HT are not restricted to the liver as this polyphenol relieves brain damage produced by subarachnoid hemorrhage in rats by preventing oxidative stress and reducing the activity of NF-κB [64], effects that are also evident in the prevention of neuronal damage induced by dopamine and 6-hydroxydopamine through an enhanced expression of phase II-detoxification enzymes such as NADPH quinone oxidoreductase 1 [65]. This evidence concerns the gene NQO1 and subarachnoid hemorrhage.