Similar to what has been reported elsewhere for DMD and animal models [27–29], up-regulated skeletal muscle genes included those associated with fibrosis (e.g., collagens, POSTN, TIMP1), inflammation (e.g., complement components, chemokines, cytokines), and muscle cytoskeletal proteins (e.g., myosin heavy chains, troponins, and actins). Here, POSTN is linked to Duchenne muscular dystrophy.