TIMP1 and Duchenne muscular dystrophy: Similar to what has been reported elsewhere for DMD and animal models [27–29], up-regulated skeletal muscle genes included those associated with fibrosis (e.g., collagens, POSTN, TIMP1), inflammation (e.g., complement components, chemokines, cytokines), and muscle cytoskeletal proteins (e.g., myosin heavy chains, troponins, and actins).