More than 100 amyloidogenic TTR point mutants are described, of which the TTR Val30Met preferentially gives rise to a sensorimotor amyloidotic polyneuropathy (TTR-FAP) [2], whereas other mutations, such as the Val122Ile, predispose to the development of cardiomyopathy (TTR-FAC) [3–8]. This evidence concerns the gene TTR and cardiomyopathy.