MYCN and medulloblastoma: Of note, amplifications and overexpression of either Myc or MycN oncogenes are relatively common among all subtypes of medulloblastoma, and Myc has been already implicated as a trigger of cellular replication stress (Gajjar and Robinson, 2014; Maya‐Mendoza et al., 2015; Northcott et al., 2012; Pui et al., 2011).