The biomarkers analyzed in this study are representative of different steps in the physiopathology of sepsis: recognition of the antigen by monocytes/macrophages (sTREM-1, sCD14), monocyte-endothelium interactions (sCD163), synthesis of proinflammatory cytokines (IL-6), and the response of target organs to these cytokines (PCT and CRP). The gene discussed is CD163; the disease is Sepsis.