The IL‐23/Th17 axis has been regarded as a key master pathway in psoriasis and also enhances the development of several other models of autoimmune disease including experimental allergic encephalomyelitis (EAE; Chen et al, 2006), colitis (Sivanesan et al, 2016), and diabetes (Bellemore et al, 2016). This evidence concerns the gene IL23A and colitis.