TRAF3IP2 and psoriasis: Increasing evidence indicates that genetic mutations in genes such as TNFAIP3, TNIP1, NFKBIA, TRAF3IP2, and CARD14 result in an impaired negative regulation of NF‐κB proinflammatory activity leading to psoriasis (Nair et al, 2009; Huffmeier et al, 2010; Strange et al, 2010; Jordan et al, 2012).