Although injecting IL‐23 might induce atopic dermatitis‐like inflammation rather than psoriasis‐like inflammation in CCR2‐deficient mice (Bromley et al, 2013), the robust Th1, Th2, Th17 activation, which is similar to human atopic dermatitis, is seen in IL‐23‐injected mice (Ewald et al, 2017). The gene discussed is IL23A; the disease is psoriasis.