These conditions lead to hepatic steatosis, over-secretion of larger triglyceride (TG)-rich very low density lipoprotein 1(VLDL1) particles into the plasma, over-secretion of hepatic apolipoprotein B (ApoB), impaired clearance of chylomicrons and decreased receptor mediated endocytosis in the liver [6, 7]. Here, APOB is linked to Hepatic steatosis.