HGF and Behcet disease: We therefore assume that not negative life events and a higher HPA-axis activity, but the earlier mentioned putative endothelial damage and consequent transition of the circulating monocytes into anti-inflammatory vessel repair cells CACs plays a overruling and prime role in the here found characteristic profile (high HGF, reduced pro-inflammatory compounds) of the circulating monocytes in the BD patients of the BiSS cohort.