Although the etiology of BWF has been the subject of considerable speculation, it has frequently been linked to prior treatment with either quinine [3] or synthetic arylamino alcohol antimalarials [4] such as mefloquine and halofantrine, or to glucose-6-phosphate dehydrogenase (G6PD) deficiency [5]. The gene discussed is G6PD; the disease is hyperinsulinemic hypoglycemia, familial, 4.