Because TSPO tracers such as [11C](R)PK11195 are considered to be PET probes of delayed microglial activation as shown in literature [22], the presence of [11C]NE40 uptake along with a lack of [11C](R)PK11195 uptake suggests that CB2 may be more implicated than TSPO during the early-phase activation of microglia in the post-stroke brain region. The gene discussed is CNR2; the disease is Stroke.