SERPINA1 and severe congenital neutropenia: The genetic complementation analyses of GmSNAP18 clearly showed that the transgenic ExF50 (GmSNAP18−/GmSHMT08+) gained resistance to SCN after being complemented with the Forrest GmSNAP18, but not when complemented with Forrest AAT (Fig. 5a and Supplementary Fig. 4), indicating that the Forrest GmSNAP18 is the gene at the rhg1-a locus that confers resistance to SCN.