By using an oncolytic poxvirus that is designed to attract T cells with high efficiency (CXCL-11 expression) and upregulate PD-L1 (IFN-γ response to poxvirus infection as one of the mechanisms), the transformation of anti-PD-L1 resistant tumours into sensitive tumours is feasible. Here, CXCL11 is linked to neoplasm.