TLR9 and infection: The study performed for Polish fetuses and neonates congenitally infected with HCMV, showed the CC genotype in TLR4 1196 polymorphism, as well as the GA variant in TLR9 2848 G > A SNP to be correlated with the infection, and the heterozygotic status in TLR9 SNP increased the risk of congenital cytomegaly by 4.81 times [18].