CLEC7A and infection: Dectin-1−/− mice presented a decreased number of parasitized (CFSE+) cells in the peritoneal cavity during the acute phase (p < 0.01, Figure 1B), and lower concentration of parasite genomic DNA during chronic infections (p = 0.0002, Figure 1C), demonstrating that the presence of functional Dectin-1 contributes to the susceptibility of mice to the infection by N. caninum.