B2M and Hyponatremia: We found that a substantial number of patients with sAKI had hyponatremia, and patients with sAKI had significantly higher fractional excretion of sodium than those without sAKI, indicating the occurrence of renal tubular damage among patients with sAKI, which is consistent with a previous study from Myanmar reporting increased levels of beta-2 microglobulin and N-acetyl glucosaminidase in urine among patients with sAKI from Russell’s viper bites, also indicating renal tubular damage [27].