In addition to CD82 and integrinβ1, IL-8, cyclooxygensase-2 (Cox-2) and MMPs may be involved in this process.7, 41 IL-27 further amplified the stimulatory effect of Th17 on the growth and invasion of ESCs in vitro, and increased the number and weight of ectopic lesions in the mouse endometriosis model. Here, CD82 is linked to endometriosis.