As one of the plausible modes of IFN-β action in responsive patients is anti-inflammatory effect (Graber et al. 2007; Liu et al. 2010; Ransohoff et al. 1991; Rio and; Montalban 2005; Salama et al. 2003), the improved results of IFN-β therapy could be especially expected from early treatment of MS, when the inflammatory process initiates. The gene discussed is IFNB1; the disease is myeloid sarcoma.