A similar effect, i.e. elevation of pro-inflammatory cytokines (including IFN-γ, IL-1β, TNF-α, and IL-6) was observed in experimental autoimmune encephalomyelitis (EAE), an animal model of MS (Schneider et al. 2009; Sulkowski et al. 2013; Tanuma et al. 1997). This evidence concerns the gene IL1B and myeloid sarcoma.