Poststreptococcal glomerulonephritis (PSGN), induced by “nephritogenic” group A streptococcal pharyngitis or skin infections, is a classic example of PIGN with diffuse proliferative and exudative glomerular histology, dominant C3 staining, some IgG deposition, and subepithelial hump-like deposits [6, 12]. This evidence concerns the gene C3 and acute proliferative glomerulonephritis.