Increasing evidence implicates involvement of ILC3s in psoriasis, a skin inflammatory disease largely resulting from overproduction of Th17-associated cytokines such as IL-17A (Dyring-Andersen et al., 2014; Pantelyushin et al., 2012; Teunissen et al., 2014; Villanova et al., 2014). This evidence concerns the gene IL17A and psoriasis.