FGF2 and Stroke: Similarly, another study has found that miR-15 is upregulated in endothelial cells up to 16 h after oxygen deprivation exposure in vitro and provided evidence suggesting that pharmacological inhibition of miR-15a may be a potential approach to increasing angiogenesis after stroke through targeting of basic fibroblast growth factor (bFGF) and VEGF in endothelial cells [51,52].