Findings in the model of infection by AIEC LF82 indicated that bLf inhibited the bacterial invasion and the pro-inflammatory cytokine response of TNFα, IL-6 and IL-8 in epithelial monolayers and in cultures from colonic biopsies from patients with Crohn’s disease, suggesting a potential therapeutic role for bLf as antibacterial and anti-inflammatory agent [104]. Here, CXCL8 is linked to infection.