Disrupted regulation of apoptosis is the hallmark of carcinogenesis allowing accumulation of further genetic mutations and acquisition of metastatic properties.1 Cancer cells often express increased levels of anti-apoptotic Bcl-2 (B-cell lymphoma-2) proteins, which provides additional protection against cell death signals2, 3 correlating with chemotherapy resistance and poor prognosis for patients.4 Here, BCL2 is linked to cancer.