AKT1 and bronchopulmonary dysplasia: It is important to note that in the current study Akt activity was not measured additionally; however, previous reports in other models point in the same direction: H2S treatment enhanced Akt phosphorylation in models of liver ischemia-reperfusion [43, 44], in lung injury due to infrarenal aortic cross-clamping [45], and bronchopulmonary dysplasia [46], suggesting a central role for Akt activation in H2S-mediated organ-protective effects.