Recently, the EGFRvIII/Ras/PI3K/AKT axis has been shown to exert its tumorigenic influence through the specific inhibition of miR‐9 leading to the up‐regulation of the transcription factor FOXP1, thus providing a tumor growth advantage to EGFRvIII‐driven tumors (Gomez et al., 2014). This evidence concerns the gene AKT1 and neoplasm.