A similar localization pattern for pSmad3, nuclear in stromal MCs but not in the tumour, was also observed in peritoneal implants of colon cancer patients, suggesting that a crosstalk mechanism via TGF‐β1–pSmad3 could be common to cancers that metastasize via peritoneal carcinomatosis (supplementary material, Figure S4). This evidence concerns the gene TGFB1 and neoplasm.