At least one reason for the suitability of this strain appeared to be its poor swimming motility, which is consistent with the observation (Table 1) that a fliI mutant of strain PA14 caused similar nonlethal, nondisseminating cutaneous abscesses and dermonecrotic lesions (cf. the parent PA14 isolate) that caused progressive mortality and frequent dissemination to the kidneys and/or liver. The gene discussed is FLII; the disease is Cutaneous abscess.