To this day, there is no cure for ADOA, but a study performed by Barboni et al. in 2013, showed for the first time an improved visual function, after application of a synthetic analogue of coenzyme Q10 (Idebenone), an antioxidant used to reduce reactive oxygen species (ROS) levels in Leber’s hereditary optic neuropathy (LHON) patients, in ADOA patients with OPA1 mutations [38]. This evidence concerns the gene OPA1 and Leber hereditary optic neuropathy.