Despite the fact that TREK‐1 is also expressed in the brain and the adrenal gland, extra‐cardiac phenotypes [e.g., a neurological disorder or primary hyperaldosteronism caused by a Na+ leak in the adrenal gland, as observed for somatic mutations in KCNJ5 (Gomez‐Sanchez & Oki, 2014; Kuppusamy et al, 2014)] were not observed in the patient. The gene discussed is KCNK2; the disease is nervous system disorder.