We have previously demonstrated that targeted cell therapy with RAECs and human iPS‐ECs overexpressing IL8RA/RB inhibits early inflammatory mediator expression, inflammatory cell infiltration, and subsequent neointima formation in a variety of models of acute vascular injury, including balloon‐injured rat carotid arteries 10, 12, monocrotalin‐induced acute lung injury 22, and coronary ligation‐induced myocardial infarction 23. Here, RB1 is linked to myocardial infarction.