In turn, using a monolayer wound healing assay siRNA-mediated knockdown of DAPK1 clearly increased tumor cell migration of ΔpBK-ITIH5 clone #7 compared to corresponding cells transfected with the non-silencing control siRNA (nc-control siRNA) that served as negative control (Fig. 9c and d). This evidence concerns the gene DAPK1 and neoplasm.