On the other hand, Met exerted antitumorigenic effect in cervical carcinoma HeLa and SiHa cells by decreasing the expression of a limiting glycolytic enzyme Pyruvate kinase muscle isozyme M2 (PKM2) involved in mTOR-dependent pathway regulating epithelial-mesenchymal transition (EMT), as reported by Cheng et al. [22]. This evidence concerns the gene MTOR and cervical carcinoma.