Interestingly, the results of the current study are consistent with a previous report that SAMC prevented liver disease by diminishing NF-κB activity andinhibiting the expression of pro-inflammatory cytokinesTNF-α, IL-1β and TGFβ1, as well as inflammatoryenzymes COX-2 and iNOS (inducible nitric oxide synthase) [25]. The gene discussed is NFKB1; the disease is liver disorder.