Numerous studies have indicated that NAFLD37, 38, 39 and H. pylori infection40, 41, 42 may cause dyslipidemia by affecting TC, TG, LDL, HDL and OxLDL levels in addition to dysapolipoproteins, including ApoA1, ApoB and the ApoA1/ApoB ratio, and that these effects may impact the incidence and development of atherosclerosis. This evidence concerns the gene APOA1 and atherosclerosis.