CAMK2G and cardiac arrhythmia: Isolated myocytes studies have shown that CaMKII activation enhances the INa-L and the late ICa,L(Xie et al., 2009; Belardinelli et al., 2015) promoting EADs and triggered arrhythmias (Morotti et al., 2014; Foteinou et al., 2015; Pezhouman et al., 2015).