MSANTD3 and acinar cell carcinoma: In summary, by whole-transcriptome sequencing of archival AcCC cases and validation by FISH, we have discovered novel gene fusions including a recurrent rearrangement of the previously uncharacterized gene MSANTD3. Functional studies implicate a role of MSANTD3 in upregulating translation; however, the normal and tumorigenic mechanisms of this novel putative oncogene await future investigation.