In the presence of follistatin, myostatin is not able to connect to its own receptor and this can prevent the muscular dystrophy caused by myostatin.8,9 Myostatin expressed during periods of inactivity increases skeletal muscle10 or the inhibition of serum myostatin increases strength and muscle mass.11 Therefore, it seems that the resistance training leads to decreased expression of myostatin.12 According to the findings, the myostatin gene expression in the heart muscle can change following the physical activity or the myocardial infarction. The gene discussed is MSTN; the disease is muscular dystrophy.