It is well known that missense mutations in coding sequences in the GABAA receptor γ2 subunit gene GABRG2 are associated with relatively mild epilepsy phenotypes, including childhood absence epilepsy and febrile seizures (Wallace et al., 2001; Audenaert et al., 2006; Shi et al., 2010), and with the genetic epilepsy with the febrile seizures plus (GEFS+) spectrum (Baulac et al., 2001). Here, GABRG2 is linked to epilepsy.