Despite similar blood antioxidant capacity, paraoxonase concentration was in fact lower in OVE cows, a condition that not only indicates liver dysfunction, but one that has been proven to lead to an increase in the inflammatory status (confirmed by higher ceruloplasmin and SAA), which notoriously causes an increase in oxidative stress, and a reduction of antioxidative protection during the early postpartum period [27, 52]. Here, SAA2 is linked to Decreased liver function.