BCL2L11 and pituitary gland adenoma: In this scenario, it seems conceivable that a specific expression profile (such as high D2 vs. sst5 expression levels in stimulated-pituitary adenomas compared to similar D2/sst5 expression levels in inhibited-pituitary adenomas) could be a key molecular determinant for the response to BIM-23A760, as it could dictate the possible interactions between receptor subtypes (homo and/or heterodimerization).