Therefore, these findings might suggest that the relative sst5 expression levels (i.e. lower sst5 levels in stimulated vs. inhibited pituitary adenomas as well as, lower sst5 as compared with D2 levels in the stimulated pituitary adenomas) might represent a potential molecular signature contributing to the differential response to BIM-23A760. Here, SSTR5 is linked to pituitary gland adenoma.