Additionally, our results show that in breast cancer epithelial cells, hATT-CMs increase: 1) membrane glycoprotein CD44 (which plays a key role in cell adhesion as well as in cell migration); 2) metalloproteinase ADAMTS1 (which is capable of proteoglycan cleavage and ECM degradation); 3) adiponectin type 1 receptor (one of adiponectin receptors). The gene discussed is ADAMTS1; the disease is breast carcinoma.