As Hif‐1α expression is impaired by high glucose in diabetes 29, the increased angiogenic capacity of G‐D‐ADSCs might be related to the elimination of ROS and decreased oxidative stress caused by GLO1 that can lead to upregulation of Hif‐1α and, consequently, elevated expression of Sdf‐1α and Vegfa. This observation might be the underlying mechanism of GLO1 overexpression‐mediated restoration of the proangiogenic capacity of D‐ADSCs. The gene discussed is CXCL12; the disease is diabetes mellitus.