While the promoters of these two clusters were shown to be p53‐responsive in several cancer lines (Braun et al., 2008; Pichiorri et al., 2010), another study reported that inhibition of p53 did not prevent the induction of miR‐194‐2/192 by aristolochic acid in proximal tubular epithelial cells (Jenkins et al., 2014), indicating that the mechanisms by which expression of miR‐194 clusters is regulated are diverse and may operate in a context‐specific way. Here, TP53 is linked to cancer.