Recently, Yoshikawa et al. (2016), using a murine model of deep dermatophytosis with lack of Dectin-1 and/or Dectin-2, demonstrated that absence of these C-type lectin receptors promotes an inefficient pro-inflammatory response against T. rubrum infection characterized by lower production of TNF-α and IL-1β by spleen cells and impairing the dermatophytosis resolution compared to control mice (Yoshikawa et al., 2016). Here, IL1B is linked to dermatophytosis.