We mapped by ChIP-seq using anti-lamin A/C antibodies, LMNA LADs in fibroblasts from four patients with familial partial lipodystrophy of Dunnigan type (FPLD2; OMIM#151160; patients “p1–p4”) bearing the same heterozygous LMNA p.R482W mutation [49] and in fibroblasts from three healthy control individuals. The gene discussed is LMNA; the disease is familial partial lipodystrophy, Dunnigan type.