However, for all breast cancer cells ID extract affected apoptosis-regulated gene products involved in pro-apoptotic (Bax) and anti-apoptotic proteins (Bcl-2 and XIAP), as well as apoptosis target (caspase-9) and DNA repair proteins (PARP), thereby suppressing the cell proliferation and inducing apoptosis. This evidence concerns the gene CASP9 and breast cancer.