RT also causes release of danger-associated molecular patterns (DAMPs) from stressed or dying tumor cells, including pro-inflammatory cytokines (e.g., TNFα, IFNγ, IL-1α/β) and other immune-stimulatory factors (e.g., ATP, HMGB1, cyclic dinucleotide, calreticulin) in what has been described as ‘immunogenic cell death’ [109,110,111]. The gene discussed is IL1A; the disease is neoplasm.